blind schizophrenia

the relevant literature includes various phrasings of this idea, ranging from milder claims about cb serving as a protective factor that reduces the risk to develop schizophrenia (silverstein et al., 2013a) to stronger claims which imply that this risk is reduced to zero, as in sanders et al. the paper is organized as follows: in section cases of congenital blindness and schizophrenia, the distinction between ccb and cpb is presented and the reasons for assuming it are discussed. starting off from stewart and sardo (1965), this is the only case of schizophrenia and cb brought up in silverstein et al. on this basis, we are inclined to assume this is a case of hereditary blindness that involves the globe (i.e., cpb). on this basis, we do not have any reason to question the diagnosis of schizophrenia that is given in kerschbaumer (1943). in other words, the genetic basis of bardet-biedl suggests that this is a syndrome that may manifest comorbidity between schizophrenia and retinitis pigmentosa.

dlg4 encodes a member of the membrane-associated guanylate kinase family and has been recently argued to play a role in schizophrenia pathogenesis (balan et al., 2013). as the presentation of different syndromes in section cases of congenital blindness and schizophrenia suggested, a link seems to exist between visual dysfunction of peripheral origin and schizophrenia. the picture is different in cases of ccb, where subcortical structures are intact and have been reported to be activated for eye contact (burra et al., 2013). the relation between schizophrenia and language has at times been approached in terms of the former being a disruption of the latter. there is a variety of studies reporting impaired plasticity, dysfunctional network connectivity, and a disturbed shape due to disrupted corticogenesis in relation to the visual cortex of schizophrenic individuals (çavuş et al., 2012; schultz et al., 2013; ford et al., 2014). on this basis, we suggested that the distinction between different types of blindness in terms of the origin of the visual deficit is crucial for understanding both the observed patterns of comorbidity and the nature of the protective effects. if all causes of cb excluding that of cortical origin have a prevalence of 82% in total and if this roughly translates to the probability of comorbid congenital peripheral blindness and schizophrenia being 0.02% according to silverstein et al.

and though there have been countless studies conducted to test the theory, none have been as comprehensive as one published in schizophrenia research in 2018, which looked at whole-population data from 467,945 children born in western australia between 1980 and 2001. over the course of the study, scientists found that of the 1,870 children (0.4 percent) who developed schizophrenia, none were born blind. “one theory might be that a person blind from birth learns to think in ways that are protective, or the brain learns to use that extra cortex in ways that are protective,” dr. sedlak says. while the most characteristic symptoms of schizophrenia are the psychotic ones such as hallucinations and delusions, scientists believe that these are not actually the core features of the disease. the belief is that these kinds of enhanced skills have a protective effect from developing the exact opposite impairments, which come with schizophrenia.

scientists believe that when someone’s vision is abnormal, the brain receives all kinds of confusing signals about the world, so they have to make more predictions in order to make sense of it all. on the other hand, scientists hypothesize that when someone is blind from birth, the brain is conditioned to make sense of all of that sensory information coming in by relying on other cues to build a mental picture. though a hard and fast explanation for the absence of schizophrenia in congenital blindness remains to be unfurled, these hypotheses are grounds for hope in understanding and treating the condition. “congenital blindness is protective for schizophrenia and other psychotic illness. “cognitive deficits in schizophrenia: understanding the biological correlates and remediation strategies” this information is not designed to replace a physician’s independent judgment about the appropriateness or risks of a procedure for a given patient.

blindness that is the result of childhood-onset retinitis pigmentosa and schizophrenia-like psychosis can co-exist in bardet-biedl syndrome (weiss et al., 1981) people with schizophrenia have been shown to have problems with their vision, including retina issues, unusual eye movements, and abnormal these findings suggest that something about congenital blindness may protect a person from schizophrenia. this is especially surprising, since, symptoms of schizophrenia, symptoms of schizophrenia, related conditions, deaf person with schizophrenia, schizophrenia vision problems.

the words above suggest that blindness may be protective of schizophrenia, and represent the first formulation of this hypothesis, published in as a paper just published in frontiers in psychology points out, congenital and early blindness appears to protect against schizophrenia. the conclusion that there are no c/e blind people with schizophrenia is based on a small number of studies that involved relatively small, no one born blind has ever developed schizophrenia source of knowledge, cortical blindness and schizophrenia, schizophrenia wiki, congenital blindness is protective for schizophrenia, schizophrenia eye symptoms, can mental illness cause blindness, congenitally blind, has a blind person ever been cured, broccoli and schizophrenia, vice schizophrenia.

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